Health
‘COVID paralyzed my diaphragm’: Marathon runner shares how the infection took his breath away
Someone with COVID-19 might expect to experience fatigue, cold and flu symptoms, and loss of taste and smell — but a paralyzed diaphragm would likely not be on the radar.
It was certainly a surprise to Gerald Branim, 55, a runner from Tennessee. His life took a drastic turn when COVID-19 damaged his lungs and diaphragm to the extent that he was unable to run or walk for a year.
Branim and his doctor, Matthew Kaufman of the Institute of Advanced Reconstruction in New Jersey, joined Fox News Digital in an interview to discuss Branim’s ordeal and recovery.
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When Branim got COVID in Feb. 2021, he was a fit, healthy 52-year-old who ran marathons.
In spite of that, the infection caused significant lung damage. He spent two weeks in the hospital and three months out of work.
Gerald Branim, 55, was a marathon runner when he got COVID, which led to a paralyzed diaphragm that stopped him in his tracks. (Institute for Advanced Reconstruction)
For two months after leaving the hospital, Branim used a walker as he continued struggling with lung complications.
“For someone who had run marathons, it was quite humbling to have to walk with a walker for three months,” he said.
After rounds of high-dose steroids and lots of antibiotics, Branim’s chest X-rays finally showed that his lungs had cleared — but he was still having trouble functioning.
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“I still couldn’t go upstairs without my oxygen dropping severely,” he recalled. “I couldn’t run 100 yards without my oxygen dropping into dangerous levels. I was severely out of breath.”
“That’s when they started saying something else was going on — that this shouldn’t be happening.”
“I couldn’t run 100 yards without my oxygen dropping into dangerous levels. I was severely out of breath.”
After 10 months of not being able to walk or run, Branim’s condition was finally diagnosed via a “sniff test,” which is also called a chest fluoroscopy or a video chest X-ray, where the patient is asked to breathe in and out.
Any paralysis will show up on the scan, as the diaphragm won’t move upon breathing.
What to know about diaphragm paralysis
Diaphragm paralysis — which is a “pretty rare condition,” according to Kaufman, Branim’s doctor — is paralysis of the diaphragm muscle, which is the primary breathing muscle.
Previous studies have estimated that the condition affects only about 1.31% of the population. In about 95% of cases, only one side is paralyzed, the doctor noted.
“We have two diaphragms, one on the right, one on the left,” he said.
Branim, far right, is pictured with his doctor, Matthew Kaufman, center, and another patient at the half-marathon they ran together after Branim’s surgery. (Institute for Advanced Reconstruction)
In cases where both sides are paralyzed, symptoms will be much more severe, usually requiring the patient to be put on oxygen.
“It’s become sort of a phenomenon,” Kaufman told Fox News Digital. “The phrenic nerve, which is the nerve that controls the diaphragm, seems to be an area where the COVID virus can have an impact.”
In some ways, he said, the condition can be considered part of long COVID, which is when symptoms of the virus linger for weeks, months or even years after infection.
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Most people with diaphragm paralysis won’t have difficulty breathing while sitting. Symptoms will typically emerge with exertion, such as walking or exercising, or when changing positions, such as lying flat in bed or bending over to tie their shoes, Kaufman said.
Some people are more susceptible than others, the doctor noted.
“We know that viruses tend to attack nerves in the body, as we’ve seen in Bell’s palsy,” he said. “And now we’re seeing it with COVID causing injury to the phrenic nerve.”
Branim set a goal of running a half-marathon within a year of his surgery date, which he achieved alongside his doctor in April 2023. (Institute for Advanced Reconstruction)
Because the condition is rare to begin with, he said, it’s not something that’s universally known.
“And then you take a condition like COVID, which causes respiratory symptoms from inflammation in the airways and lungs, and then you tie in what we consider to be a neuromuscular problem,” Kaufman said.
“A lot of times, the physician or patient will attribute it to airway inflammation, when in fact it’s a problem related to nerves and muscles that are part of the respiratory system.”
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In some cases, patients will recover on their own, Kaufman said.
“We usually recommend a period of about six to 12 months of physical therapy, breathing exercises and cardiovascular fitness to try to see if this will come back on its own,” he said.
If improvement is not noticed in that time frame, surgical intervention becomes a possibility.
Branim emphasized the importance of patients serving as their own advocates. “Doctors are human — they don’t know everything,” he said. “So you have to advocate for yourself.” (Institute for Advanced Reconstruction)
In 2020 and 2021, Kaufman said he saw an uptick of patients who experienced diaphragm paralysis after having COVID. Although he is still seeing some cases, they are starting to trail off a bit.
“That could be because the classic COVID is not as virulent or severe,” he said. “Or maybe it’s because more people are immunized.”
Although the condition is generally not life-threatening, it can be more severe in patients who have existing respiratory conditions.
Racing toward recovery
After Branim’s diagnosis, his lung doctor advised him to continue to give it time to heal.
“But it wasn’t getting any better,” he said. “And I just wasn’t satisfied with that being my new quality of life.”
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That’s when Branim turned to Google to research surgical options. He came across an article by Kaufman, a plastic and reconstructive surgeon who specializes in an area called peripheral nerve microsurgery.
Kaufman specializes in conditions that cause either pain or paralysis due to nerve damage of the peripheral nervous system.
After meeting with Kaufman in a telehealth visit, Branim was identified as a prime candidate for the surgery, which is called phrenic nerve reconstruction.
“It wasn’t getting any better — and I just wasn’t satisfied with that being my new quality of life.”
If he had waited much longer, he said, his diaphragm muscle would have atrophied and the surgery would no longer have been possible.
After a lot of back and forth with the insurance company, Branim finally got the green light to travel from Nashville to New Jersey for the procedure.
The surgery went smoothly, although Branim was told that it wouldn’t be an immediate improvement. Once the nerve is fixed, the muscle still needs time to strengthen and rehabilitate.
Studies have estimated that the condition affects only about 1.31% of the population. In about 95% of cases, only one side is paralyzed. (Institute for Advanced Reconstruction)
“Dr. Kaufman told me that it would probably take a year or two to recover fully, and was preaching patience — but I’m not a patient man in the slightest sense of the word,” Branim laughed.
He set a goal of running a half-marathon within a year of his surgery date.
After four weeks, Branim slowly began exercising again. After six months, he decided to lace up his running shoes and give it a try.
“My very first run after the surgery, I ran five miles,” he said. “I literally cried. At that point, it was like a light switch had been flipped.”
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Within six months, Branim had not only regained his ability to run, but exceeded his original goals, culminating in a half-marathon in April 2023.
In a triumphant twist, Kaufman, who is also a runner, joined his patient in completing the race in Asbury Park, New Jersey.
Now, Branim aims to raise awareness about diaphragm paralysis, an often-overlooked condition that is not on most doctors’ or patients’ radars.
Previous studies have estimated that the condition affects only about 1.31% of the population. In about 95% of cases, only one side is paralyzed. (iStock)
“None of my doctors had even heard of this surgery,” he said. “Had I not found the article about Dr. Kaufman and another runner online, I would have never been able to have the surgery — and I would absolutely not be running today.”
Branim emphasized the importance of patients serving as their own advocates.
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“Doctors are human — they don’t know everything,” he said. “So you have to advocate for yourself.”
For patients who are experiencing shortness of breath with exertion that persists for several months, Kaufman recommends visiting a primary care physician or pulmonary physician to get the necessary testing.
“While most tests will find more common ailments, it definitely makes sense to keep a paralyzed diaphragm on the list of things to rule out.”
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A drug that has long been used to treat seizures has shown promise as a potential means of Alzheimer’s prevention, a new study suggests.
The anti-seizure medication, levetiracetam, was first approved by the FDA in November 1999 under the brand name Keppra as a therapy for partial-onset seizures in adults. The approval has since expanded to include children and other types of seizures.
Northwestern University researchers recently found that levetiracetam prevented the formation of toxic amyloid beta peptides, which are small protein fragments in the brain that are commonly seen in Alzheimer’s patients.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons, according to the study findings, which were published in Science Translational Medicine.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons. (iStock)
“While many of the Alzheimer’s drugs currently on the market, such as lecanemab and donanemab, are approved to clear existing amyloid plaques, we’ve identified this mechanism that prevents the production of the amyloid‑beta 42 peptides and amyloid plaques,” said corresponding author Jeffrey Savas, associate professor of behavioral neurology at Northwestern University Feinberg School of Medicine, in a press release.
“Our new results uncovered new biology while also opening doors for new drug targets.”
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The brain is better able to avoid the pathway that produces toxic amyloid‑beta 42 proteins in younger years, but the aging process gradually weakens that ability, Savas noted.
“This is not a statement of disease; this is just a part of aging. But in brains developing Alzheimer’s, too many neurons go astray, and that’s when you get amyloid-beta 42 production,” he said.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease. (iStock)
That then leads to tau (“tangles”) — abnormal clumps of protein inside brain neurons — which can kill brain cells, trigger neuroinflammation and lead to dementia.
In order for levetiracetam to function as an Alzheimer’s blocker, high-risk patients would have to start taking it “very, very early,” Savas said — up to 20 years before elevated amyloid-beta 42 levels would be detected.
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“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death,” the researcher noted.
The researchers also did a deep dive into previous human clinical data to determine whether Alzheimer’s patients who were taking the anti-seizure drug had slower cognitive decline. They reported that the patients in that category had a “significant delay” in the span from cognitive decline to death compared to those not taking the drug.
“This analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” the researcher said. (iStock)
“Although the magnitude of change was small (on the scale of a few years), this analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” Savas said.
Looking ahead, the research team aims to find people who have genetic forms of Alzheimer’s to participate in testing, Savas said.
Limitations and caveats
The study had several limitations, including that it relied on animal models and cultured cells, with no human trials conducted.
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Because the study was observational in nature, it can’t prove that the medication caused the prevention of the toxic brain proteins, the researchers acknowledged.
Savas noted that levetiracetam “is not perfect,” cautioning that it breaks down in the body very quickly.
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The team is currently working to create a “better version” that would last longer in the body and “better target the mechanism that prevents the production of the plaques.”
“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death.”
The medication’s common documented side effects include drowsiness, weakness, dizziness, irritability, headache, loss of appetite and nasal congestion.
It has also been linked to potential mood and behavior changes, including anxiety, depression, agitation and aggression, according to the prescribing information. In rare cases, it could lead to severe allergic reactions, skin reactions, blood disorders and suicidal ideation.
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Funding for the study was provided by the National Institutes of Health and the Cure Alzheimer’s Fund.
Fox News Digital reached out to the drug manufacturer and the researchers for comment.
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Older adults who avoid meat in their golden years may be less likely to reach age 100 than their meat-eating counterparts, new research suggests.
Researchers tracked more than 5,000 adults aged 80 or older who were enrolled in the Chinese Longitudinal Healthy Longevity Survey.
Between 1998 and 2018, data showed that those who did not eat meat were less likely to reach their 100th birthday than those who consumed animal products regularly.
The findings seem to contradict previous studies that have linked vegetarianism and plant-based diets to lower risks of heart disease, stroke, diabetes and obesity.
Most evidence supporting the benefits of plant-based diets comes from studies tracking younger populations, the researchers noted.
The findings contrast with previous research praising plant-based diets for their positive influence on heart health. (iStock)
The study, published in The American Journal of Clinical Nutrition, points to losses in muscle mass and bone density with age, shifts that can increase the risk of malnutrition and frailty in the “oldest old.”
As people enter their 80s and 90s, the nutritional priority often shifts from preventing long-term chronic diseases to maintaining day-to-day physical function, experts say.
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“The headline ‘vegetarians over 80 less likely to reach 100’ sounds surprising, because it contrasts with decades of data linking plant‑forward diets to lower chronic disease risk earlier in life,” Erin Palinski-Wade, a New Jersey-based registered dietitian, told Fox News Digital.
“However, once you see that this research is limited to adults over the age of 80 who are also underweight — and that this link disappears with the consumption of eggs, dairy and fish — the results are less surprising.”
While diets earlier in life tend to emphasize avoiding long-term disease, older age necessitates nutrients and weight maintenance, experts say. (iStock)
In those over 80, restricting animal proteins may be less likely to promote longevity, according to Palinski-Wade, who was not involved in the study.
Eliminating all animal protein — particularly in a population that may already experience diminished hunger cues — can make it more difficult to meet adequate protein needs, potentially increasing the risk of nutrient deficiencies, the nutritionist said.
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In addition to a higher tendency to be underweight, older populations also face a greater risk of bone fractures due to lower calcium and protein intake.
Potential limitations
The lower rate of vegetarians reaching 100 was only observed in participants identified as underweight, the researchers noted. No such association was found in people who maintained a healthy weight.
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Because being underweight is already linked to greater frailty and mortality risk, the researchers noted that body weight may partly explain the findings, making it difficult to determine whether diet itself played a direct role.
Those incorporating animal-sourced products other than meat were just as likely to live to 100. (iStock)
Additionally, the shortened lifespans were not found in people who continued to eat non-meat animal products, such as fish, dairy and eggs.
Older adults with these more flexible diets were just as likely to live to 100 as those eating meat, as these foods may provide the nutrients necessary for maintaining muscle and bone health, the researchers noted.
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“This is an observational study, so it can only show associations, and does not prove that avoiding meat directly reduces the odds of reaching 100,” Palinski-Wade added.
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The researchers suggested that including small amounts of animal-sourced foods could help older seniors maintain essential nutrients and avoid the muscle loss often seen in those who stick strictly to plants.
Eliminating all animal protein — particularly in a population that may already experience diminished hunger cues — can make it more difficult to meet adequate protein needs, potentially increasing the risk of nutrient deficiencies. (iStock)
Palinski-Wade offered some guidance for those looking to optimize nutrition later in life.
“For adults in their 80s and beyond, especially anyone losing weight or muscle, the priority should be maintaining a healthy weight and meeting protein and micronutrient needs — even if that means adding or increasing fish, eggs, dairy or well‑planned, fortified plant proteins and supplements.”
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Strict vegan or very low‑protein patterns at that age should be carefully monitored by a dietitian or clinician, with attention to B12, vitamin D, calcium and total protein, according to Palinski-Wade.
“Younger and healthier adults can still confidently use plant‑forward or vegetarian patterns to lower long‑term chronic disease risk,” she added.
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