Health
Americans need more sleep, less stress, experts say, as Gallup poll reveals troubling findings
Many Americans are getting too little sleep and have too much stress.
A new Gallup poll revealed 57% of adults would “feel better if they got more sleep,” while 42% said they get “as much sleep as they need.”
These findings have nearly reversed in the last decade, Gallup noted in a press release. The last measurement in 2013 found that 56% of Americans got the sleep they needed while 43% did not.
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Overall, however, Americans are getting fewer hours of sleep than they did in past decades.
In 1942, 59% of Americans were getting eight hours or more of sleep per night, while only 3% were getting five hours or less.
Fifty-seven percent of adults said they would “feel better if they got more sleep,” a new Gallup poll revealed. (iStock)
In 2024, only 25% of Americans get an average of eight hours of sleep, and 20% reported sleeping for five or less.
Young women are the least likely to get enough sleep, according to the study — with 36% of females versus 48% of males reporting getting enough shuteye.
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Sleep amounts for both men and women showed “significant declines from previous readings in 2013 and 2004,” according to Gallup — and are the lowest measured for each group to date.
The decline was found across all age groups, although young adults between ages 18 and 29 saw the smallest difference.
Stress-sleep connection
Gallup suggested that an uptick in stress could be driving this downward trend in sleep, as the American Psychological Association reports a “strong connection between stress and sleep quality.”
The poll showed that 63% of Americans who reported wanting more sleep also “frequently experience stress.”
Women are most likely to frequently experience stress, the Gallup poll found. (iStock)
“Over the past 30 years, the number of Americans who are stressed has been on a steady incline after a sharp drop in 2003,” Gallup reported.
“The most recent data show that nearly half of all Americans, 49%, report frequently experiencing stress — up 16 points over the past two decades and the highest in Gallup’s trend to date.”
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Young women are also most likely to frequently experience stress, “exceeding men their age by 14 points,” according to Gallup.
Dr. Marc Siegel, clinical professor of medicine at NYU Langone Medical Center and a Fox News medical contributor, confirmed this relationship between sleep and stress, calling it the “cycle of worry” during a Thursday appearance on “America’s Newsroom.”
Dr. Marc Siegel, clinical professor of medicine at NYU Langone Medical Center and a Fox News medical contributor, discussed the relationship between sleep and stress during a Thursday appearance on “America’s Newsroom.” He noted that exposure to the blue light of smartphone screens can keep people awake, among other issues. (Fox News)
“They’re connected,” he said.
“If you get more stressed, you don’t sleep; if you don’t sleep, you get more stressed.”
Siegel explained that “all of this spirals out of control,” since sleeplessness is often remedied with caffeine — yet caffeine “interferes with your sleep cycle.”
“If you get more stressed, you don’t sleep; if you don’t sleep, you get more stressed.”
The same goes for drinking alcohol before bed to induce sleep, which “wears off and you wake up in the middle of the night,” the doctor warned.
Exposure to the blue light of smartphone screens can keep people awake, Siegel said.
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“All of this is very bad for health,” he said. “It leads to heart disease, it increases your risk of stroke, it causes you to gain weight.”
For young women in particular, several factors could be causing them to lose sleep, including the use of social media, which can “feed anxiety,” Siegel said.
A potential fix to ending the sleep-stress cycle is practicing “sleep hygiene,” said one doctor, which includes sleeping in a dark room away from your cellphone. (iStock)
A potential fix for the sleep-stress cycle is practicing “sleep hygiene,” Siegel suggested, which includes sleeping in a dark room away from your cell phone.
“I treat stress and sleeplessness as the same thing,” he said. “That’s why I don’t believe in sleeping pills … You’re just covering up the problem.”
He added, “I want to get at why you’re worried and what I can do about the worry.”
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Dr. Wendy Troxel, a Utah-based sleep expert and senior behavioral scientist at the RAND Corporation, told Fox News Digital in an interview that stress levels have remained “very high” since the COVID pandemic.
“[For] populations navigating multiple demands, including young people who are going to school or starting new jobs in this topsy-turvy world, it’s understandable that they are experiencing increases in stress, and that’s manifesting increases in sleep disturbances,” she said.
Dr. Wendy Troxel, senior behavioral scientist at RAND Corporation, is also the author of “Sharing the Covers: Every Couple’s Guide to Better Sleep” and scientific advisor for sleepfoundation.org. “As a culture,” she said, “we’ve become more aware of the importance of sleep over the past 10 years, which is a great thing.” (Diane Baldwin)
In some instances, Troxel pointed out, lack of sleep has been worn as a “badge of honor” to prove that people are busy or productive.
“But I think that that cultural misconception is starting to wane,” she said.
“The reality is, as a culture, we’ve just become more aware of the importance of sleep over the past 10 years, which is a great thing.”
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To break the “vicious cycle” of stress impacting sleep and vice versa, Troxel offered several tips, including maintaining a consistent sleep and wake schedule to ensure that stress doesn’t “invade your life.”
Incorporating a wind-down routine prior to bed can also bring down stress levels, the sleep expert noted.
These routines can involve relaxing activities such as deep breathing exercises, cuddling with a partner, journaling, doing gentle yoga or listening to music.
Wind-down activities before bed can include deep breathing exercises, cuddling with a partner, journaling, doing gentle yoga or listening to music. (iStock)
“It’s just about finding something that you can ritualize and do on a nightly basis to set the stage … to put aside all the demands and stress of the day and prepare for winding down and [going] to sleep,” Troxel said.
For people who wake up in the middle of the night due to stress, she advised getting out of bed, performing a wind-down activity and then returning to bed.
This technique, called stimulus control, prevents the brain from forming the habit of waking up at a certain time to ruminate on stressful thoughts.
“We all have occasional stress-related sleep disturbances, but if that starts happening night after night, it becomes habit-forming,” she said.
“And that’s where we see more chronic problems like insomnia. So, if you see that happening, treat it as a habit that your brain is learning — and break it.”
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A new study suggests that middle-aged men may be more vulnerable to faster biological aging, potentially linked to exposure to “forever chemicals.”
The research, published in the journal Frontiers in Aging, examined how perfluoroalkyl and polyfluoroalkyl substances, more commonly known as PFAS, could impact aging at the cellular level.
PFAS are synthetic chemicals commonly used in nonstick cookware, food packaging, water-resistant fabrics and other consumer products, the study noted.
Their chemical structure makes them highly resistant to breaking down, allowing them to accumulate in water, soil and the human body.
Chinese researchers analyzed blood samples from 326 adults enrolled in the U.S. National Health and Nutrition Examination Survey between 1999 and 2000.
A new study suggests that middle-aged men could face accelerated biological aging at the cellular level due to exposure to PFAS. (iStock)
The researchers measured levels of 11 PFAS compounds in participants’ blood and used DNA-based “epigenetic clocks” — tools that analyze chemical changes to DNA to estimate biological age — to determine how quickly their bodies were aging at the cellular level, the study stated.
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Two compounds, perfluorononanoic acid (PFNA) and perfluorooctanesulfonamide (PFOSA), were detected in 95% of participants.
Higher concentrations of those chemicals were associated with faster biological aging in men of certain age groups, but not in women.
“People should not panic.”
The compounds most strongly linked to accelerated aging were not the PFAS chemicals that typically receive the most public attention, the researchers noted.
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“The associations were strongest in adults aged 50 to 64, particularly in men,” Dr. Xiangwei Li, professor at Shanghai Jiao Tong University School of Medicine and the study’s corresponding author, told Fox News Digital.
“While this does not establish that PFAS cause aging, it suggests that these widely present ‘forever chemicals’ may be linked to molecular changes related to long-term health and aging.”
The study found that two of the compounds were detected in 95% of participants, and higher levels were linked to faster biological aging in men ages 50–64. (iStock)
Midlife may represent a more sensitive biological period, when the body becomes more vulnerable to age-related stressors, according to the researchers.
Lifestyle factors, such as smoking, may influence biological aging markers, potentially increasing vulnerability to environmental pollutants.
While Li said “people should not panic,” she does recommend looking for reasonable ways to reduce exposure.
That might mean checking local drinking water reports, using certified water filters designed to reduce PFAS, and limiting the use of stain- or grease-resistant products when alternatives are available.
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Meaningful reductions in PFAS exposure will likely depend on broader regulatory action and environmental cleanup efforts, Li added.
The researchers noted that midlife could be a particularly sensitive stage, when the body is more susceptible to stressors associated with aging. (iStock)
Study limitations
The researchers outlined several important limitations of the research, including that the findings show an association, but do not prove that PFAS directly causes accelerated aging.
“The study is cross-sectional, meaning exposure and aging markers were measured at the same time, so we cannot determine causality,” Li told Fox News Digital.
The study was also relatively small, limited to 326 adults age 50 or older, which means the findings may not apply to younger people or broader populations.
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Researchers measured PFAS levels using data collected between 1999 and 2000, and today’s exposure patterns may differ.
Li added that while PFAS is known to persist in the environment and the body, these results should be validated through larger, more recent studies that follow participants over time.
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A drug that has long been used to treat seizures has shown promise as a potential means of Alzheimer’s prevention, a new study suggests.
The anti-seizure medication, levetiracetam, was first approved by the FDA in November 1999 under the brand name Keppra as a therapy for partial-onset seizures in adults. The approval has since expanded to include children and other types of seizures.
Northwestern University researchers recently found that levetiracetam prevented the formation of toxic amyloid beta peptides, which are small protein fragments in the brain that are commonly seen in Alzheimer’s patients.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons, according to the study findings, which were published in Science Translational Medicine.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons. (iStock)
“While many of the Alzheimer’s drugs currently on the market, such as lecanemab and donanemab, are approved to clear existing amyloid plaques, we’ve identified this mechanism that prevents the production of the amyloid‑beta 42 peptides and amyloid plaques,” said corresponding author Jeffrey Savas, associate professor of behavioral neurology at Northwestern University Feinberg School of Medicine, in a press release.
“Our new results uncovered new biology while also opening doors for new drug targets.”
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The brain is better able to avoid the pathway that produces toxic amyloid‑beta 42 proteins in younger years, but the aging process gradually weakens that ability, Savas noted.
“This is not a statement of disease; this is just a part of aging. But in brains developing Alzheimer’s, too many neurons go astray, and that’s when you get amyloid-beta 42 production,” he said.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease. (iStock)
That then leads to tau (“tangles”) — abnormal clumps of protein inside brain neurons — which can kill brain cells, trigger neuroinflammation and lead to dementia.
In order for levetiracetam to function as an Alzheimer’s blocker, high-risk patients would have to start taking it “very, very early,” Savas said — up to 20 years before elevated amyloid-beta 42 levels would be detected.
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“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death,” the researcher noted.
The researchers also did a deep dive into previous human clinical data to determine whether Alzheimer’s patients who were taking the anti-seizure drug had slower cognitive decline. They reported that the patients in that category had a “significant delay” in the span from cognitive decline to death compared to those not taking the drug.
“This analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” the researcher said. (iStock)
“Although the magnitude of change was small (on the scale of a few years), this analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” Savas said.
Looking ahead, the research team aims to find people who have genetic forms of Alzheimer’s to participate in testing, Savas said.
Limitations and caveats
The study had several limitations, including that it relied on animal models and cultured cells, with no human trials conducted.
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Because the study was observational in nature, it can’t prove that the medication caused the prevention of the toxic brain proteins, the researchers acknowledged.
Savas noted that levetiracetam “is not perfect,” cautioning that it breaks down in the body very quickly.
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The team is currently working to create a “better version” that would last longer in the body and “better target the mechanism that prevents the production of the plaques.”
“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death.”
The medication’s common documented side effects include drowsiness, weakness, dizziness, irritability, headache, loss of appetite and nasal congestion.
It has also been linked to potential mood and behavior changes, including anxiety, depression, agitation and aggression, according to the prescribing information. In rare cases, it could lead to severe allergic reactions, skin reactions, blood disorders and suicidal ideation.
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Funding for the study was provided by the National Institutes of Health and the Cure Alzheimer’s Fund.
Fox News Digital reached out to the drug manufacturer and the researchers for comment.
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