Health
Tony Robbins reveals his personal passion for feeding hungry people: 'Shows that strangers care'
FIRST ON FOX — Speaking exclusively to Fox News Digital, bestselling author and life strategist Tony Robbins is announcing on Tuesday that he and his partners in The 100 Billion Meals Challenge have secured commitments “to provide an impressive 30 billion meals to combat global hunger” in just two years of the program’s existence, he said.
The announcement comes on Giving Tuesday — significant because Robbins has pledged to match most donations that come in, whether from individuals or groups.
“If, on Giving Tuesday, someone wants to donate a dollar up to $2 million, I’ll match it,” said Robbins. “So you’ll get to double the amount of impact if you’d like to participate.”
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Robbins launched his anti-hunger initiative along with David Beasley, former head of the World Food Programme, with a goal to provide 100 billion meals across 10 years — and the program is now pacing well ahead of plans, he said.
Robbins revealed in some detail his personal connection to the issue of hunger in America and all over the world. (See the video at the top of this article.)
“I grew up in a pretty tough environment. We didn’t have any money,” said Robbins, who today is based in Florida with his wife, Sage, and their children.
Tony Robbins, bestselling author, motivational speaker, and business and life strategist, talked to Fox News Digital this week about his bold initiative to feed hungry people in America and all over the world (100billionmeals.org). (Fox News Digital )
“I had four different fathers, and they’re all good men, but they all lost their jobs at various points. And I had a Thanksgiving when I was 11 years old where there was no money and no food in the home — no food. We had saltine crackers and peanut butter. But, you know, it was Thanksgiving.”
He said his parents were also arguing at that time — “yelling at each other or blaming each other. And I have a younger brother and younger sister, five and seven years younger, and I’m trying to make sure they don’t hear,” said Robbins, describing his life as a child.
“We had saltine crackers and peanut butter. But, you know, it was Thanksgiving.”
And then, he said, “there’s a knock at the door.”
And “long story short, this guy is holding two giant bags of groceries, and he had a pan with an uncooked frozen turkey on the ground [beside him]. He said, ‘Is your father here?’ And I was like, ‘Just one moment.’”
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Robbins said the stranger was offering his family an amazing Thanksgiving meal simply out of the kindness of his heart — and as a boy from a family with very little, he said he felt “so excited,” said Robbins.
“I thought, ‘This is going to be the most magical thing.’”
His dad, however, “did not have a positive reaction” to the offer at the door.
“He looked at this man and said, ‘We don’t accept charity.’”
There was an uncomfortable exchange as his father attempted to close the door — and finally there came “a moment I’ll never forget,” said Robbins.
“The man said, ‘Sir, please don’t make your family suffer because of your ego.’
“My dad turned bright red,” said Robbins. “I remember [seeing] the veins on the side of his neck. I thought, ‘He’s going to punch the guy in the face.’ But he just dropped his shoulders. He took the food and set it down.”
“I believed that strangers cared about me and my family. And then I wanted to care about strangers.”
Said Robbins, “And I was excited. Up until that moment, I couldn’t understand what was going on with [my father], but he didn’t take care of his family. And he left our family a few days later. It was one of the roughest moments in my life — but it was also the best moment because there was food.”
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From that experience as a boy, he said, “I developed a different belief. I believed that strangers cared — and that strangers cared about me and my family. And then I wanted to care about strangers.”
And so, said Robbins, he “set a goal that when I was older, when I was 17, I’d feed at least two families — and the next year I did four and then eight.”
Once he was in business and was able to help others on a larger scale, “we got into 50 and 100 — and then 100,000. Then finally we got it up to two million people through my foundation and two million through my wife and me.”
Said Robbins about his initiative and his motivation for helping others, “When you’ve suffered this much, you don’t want anybody else to suffer.” (Fox News Digital)
From there, over time, he dramatically increased his numbers and his connection with partners in the endeavor.
Together with Beasley, formerly of the World Food Programme, “we put together this project. And the National Pasta Association, International Pasta, Feed My Starving Children, Manna Nutrition, the government of Dubai — all of them are participating. And Global Citizen is giving us a platform to reach more people. So it’s not just me. But together, what we can do is amazing.”
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He said that no child should have to die of hunger anywhere in the world.
And the same goes for “here in America,” Robbins emphasized. “I’m doing another billion meals in America. We are the richest country in the world, and yet we still have roughly 40 million people, a lot of them children and elderly, that are still food insecure. It’s crazy. We need to do our part.”
(Robbins, with his wife, Sage Robbins, beside him, travels all over the world for his anti-hunger initiative. )
Global hunger has risen sharply in recent years — with acute hunger surging to 730 million people following the pandemic, according to Robbins and his team.
Additionally, some 30 million people “are on the brink of famine.”
“I think I suffered that way so that others wouldn’t otherwise.”
Robbins told Fox News Digital, “I think if I hadn’t grown up hungry, if I hadn’t suffered, I don’t think I would work this hard. But when you’ve suffered this much, you don’t want anybody else to suffer. And when I go to various parts of the world, including in this country where people are food insecure, I see that they don’t have any food.”
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He said these up-close visuals and experiences “almost bring me to tears. It’s just a horrific thing to see a child who’s starving in a world that’s so abundant. And once you’ve seen those images in real life, not some picture [somewhere], you can’t put it out of your mind.”
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Robbins noted, “I always say there are two types of motivation in life. There’s push motivation, when you’re trying to make yourself do something. And then there’s pull motivation — where it’s a calling, where you feel like, by God’s grace or the universe’s grace, whatever you believe in, that you’re meant to do this. And I think I suffered that way so that others wouldn’t otherwise.”
He said this is why “it’s not hard to keep your passion going, especially when you see the joy in people’s eyes when that food is brought to them. Because it’s more than food. For me, it’s that people care. People are no longer alone. And I think we can all play that role.”
Said Robbins to Fox News Digital, “Together, we can do just about anything.” Anyone can learn more about his anti-hunger efforts at 100billionmeals.org. (Carlo Allegri For The Washington Post via Getty Images)
Robbins noted the health benefits of giving to others.
“There are all kinds of studies to show that [helping others] creates a biochemical change in you. Something as simple as standing in line at Starbucks and paying for the coffee of the next five people — this will produce a larger chemical change in your body that lasts longer than usually getting something for yourself. It’s pretty wild.”
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He said, “As human beings, the reason we survive is because of our connection to community. We can never make it on our own — but together we can do just about anything.”
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A new study suggests that middle-aged men may be more vulnerable to faster biological aging, potentially linked to exposure to “forever chemicals.”
The research, published in the journal Frontiers in Aging, examined how perfluoroalkyl and polyfluoroalkyl substances, more commonly known as PFAS, could impact aging at the cellular level.
PFAS are synthetic chemicals commonly used in nonstick cookware, food packaging, water-resistant fabrics and other consumer products, the study noted.
Their chemical structure makes them highly resistant to breaking down, allowing them to accumulate in water, soil and the human body.
Chinese researchers analyzed blood samples from 326 adults enrolled in the U.S. National Health and Nutrition Examination Survey between 1999 and 2000.
A new study suggests that middle-aged men could face accelerated biological aging at the cellular level due to exposure to PFAS. (iStock)
The researchers measured levels of 11 PFAS compounds in participants’ blood and used DNA-based “epigenetic clocks” — tools that analyze chemical changes to DNA to estimate biological age — to determine how quickly their bodies were aging at the cellular level, the study stated.
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Two compounds, perfluorononanoic acid (PFNA) and perfluorooctanesulfonamide (PFOSA), were detected in 95% of participants.
Higher concentrations of those chemicals were associated with faster biological aging in men of certain age groups, but not in women.
“People should not panic.”
The compounds most strongly linked to accelerated aging were not the PFAS chemicals that typically receive the most public attention, the researchers noted.
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“The associations were strongest in adults aged 50 to 64, particularly in men,” Dr. Xiangwei Li, professor at Shanghai Jiao Tong University School of Medicine and the study’s corresponding author, told Fox News Digital.
“While this does not establish that PFAS cause aging, it suggests that these widely present ‘forever chemicals’ may be linked to molecular changes related to long-term health and aging.”
The study found that two of the compounds were detected in 95% of participants, and higher levels were linked to faster biological aging in men ages 50–64. (iStock)
Midlife may represent a more sensitive biological period, when the body becomes more vulnerable to age-related stressors, according to the researchers.
Lifestyle factors, such as smoking, may influence biological aging markers, potentially increasing vulnerability to environmental pollutants.
While Li said “people should not panic,” she does recommend looking for reasonable ways to reduce exposure.
That might mean checking local drinking water reports, using certified water filters designed to reduce PFAS, and limiting the use of stain- or grease-resistant products when alternatives are available.
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Meaningful reductions in PFAS exposure will likely depend on broader regulatory action and environmental cleanup efforts, Li added.
The researchers noted that midlife could be a particularly sensitive stage, when the body is more susceptible to stressors associated with aging. (iStock)
Study limitations
The researchers outlined several important limitations of the research, including that the findings show an association, but do not prove that PFAS directly causes accelerated aging.
“The study is cross-sectional, meaning exposure and aging markers were measured at the same time, so we cannot determine causality,” Li told Fox News Digital.
The study was also relatively small, limited to 326 adults age 50 or older, which means the findings may not apply to younger people or broader populations.
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Researchers measured PFAS levels using data collected between 1999 and 2000, and today’s exposure patterns may differ.
Li added that while PFAS is known to persist in the environment and the body, these results should be validated through larger, more recent studies that follow participants over time.
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A drug that has long been used to treat seizures has shown promise as a potential means of Alzheimer’s prevention, a new study suggests.
The anti-seizure medication, levetiracetam, was first approved by the FDA in November 1999 under the brand name Keppra as a therapy for partial-onset seizures in adults. The approval has since expanded to include children and other types of seizures.
Northwestern University researchers recently found that levetiracetam prevented the formation of toxic amyloid beta peptides, which are small protein fragments in the brain that are commonly seen in Alzheimer’s patients.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons, according to the study findings, which were published in Science Translational Medicine.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease.
The medication was found to prevent the formation of amyloid-beta 42 in both animal models and cultured human neurons. (iStock)
“While many of the Alzheimer’s drugs currently on the market, such as lecanemab and donanemab, are approved to clear existing amyloid plaques, we’ve identified this mechanism that prevents the production of the amyloid‑beta 42 peptides and amyloid plaques,” said corresponding author Jeffrey Savas, associate professor of behavioral neurology at Northwestern University Feinberg School of Medicine, in a press release.
“Our new results uncovered new biology while also opening doors for new drug targets.”
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The brain is better able to avoid the pathway that produces toxic amyloid‑beta 42 proteins in younger years, but the aging process gradually weakens that ability, Savas noted.
“This is not a statement of disease; this is just a part of aging. But in brains developing Alzheimer’s, too many neurons go astray, and that’s when you get amyloid-beta 42 production,” he said.
The effect was also seen in post-mortem human brain tissue obtained from individuals with Down syndrome, who are at high risk for Alzheimer’s disease. (iStock)
That then leads to tau (“tangles”) — abnormal clumps of protein inside brain neurons — which can kill brain cells, trigger neuroinflammation and lead to dementia.
In order for levetiracetam to function as an Alzheimer’s blocker, high-risk patients would have to start taking it “very, very early,” Savas said — up to 20 years before elevated amyloid-beta 42 levels would be detected.
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“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death,” the researcher noted.
The researchers also did a deep dive into previous human clinical data to determine whether Alzheimer’s patients who were taking the anti-seizure drug had slower cognitive decline. They reported that the patients in that category had a “significant delay” in the span from cognitive decline to death compared to those not taking the drug.
“This analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” the researcher said. (iStock)
“Although the magnitude of change was small (on the scale of a few years), this analysis supports the positive effect of levetiracetam to slow the progression of Alzheimer’s pathology,” Savas said.
Looking ahead, the research team aims to find people who have genetic forms of Alzheimer’s to participate in testing, Savas said.
Limitations and caveats
The study had several limitations, including that it relied on animal models and cultured cells, with no human trials conducted.
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Because the study was observational in nature, it can’t prove that the medication caused the prevention of the toxic brain proteins, the researchers acknowledged.
Savas noted that levetiracetam “is not perfect,” cautioning that it breaks down in the body very quickly.
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The team is currently working to create a “better version” that would last longer in the body and “better target the mechanism that prevents the production of the plaques.”
“You couldn’t take this when you already have dementia, because the brain has already undergone a number of irreversible changes and a lot of cell death.”
The medication’s common documented side effects include drowsiness, weakness, dizziness, irritability, headache, loss of appetite and nasal congestion.
It has also been linked to potential mood and behavior changes, including anxiety, depression, agitation and aggression, according to the prescribing information. In rare cases, it could lead to severe allergic reactions, skin reactions, blood disorders and suicidal ideation.
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Funding for the study was provided by the National Institutes of Health and the Cure Alzheimer’s Fund.
Fox News Digital reached out to the drug manufacturer and the researchers for comment.
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