Health
Feeling extra tired? This virus could be the culprit, study suggests
People who have had COVID-19 are more likely to develop chronic fatigue syndrome, officially known as myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS).
That’s according to a study published in the Journal of General Internal Medicine earlier this month, which found that 4.5% of people infected with SARS-CoV-2, the virus that causes COVID, developed chronic fatigue.
In comparison, only 0.6% of study participants who did not have COVID developed the second condition.
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The researchers determined that cases of ME/CFS were 15 times higher than pre-pandemic levels.
People who have had COVID-19 are more likely to develop chronic fatigue syndrome, officially known as myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS). (iStock)
“As a researcher dedicated to understanding how viral infections lead to chronic illness, I suspected from the beginning of the pandemic that SARS-CoV2 could trigger ME/CFS, so these results are not surprising,” said lead study author Suzanne D. Vernon, PhD, from the Bateman Horne Center in Salt Lake City.
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The study was part of the National Institute of Health’s RECOVER (Researching COVID to Enhance Recovery) program, which aims to better understand, prevent and treat long COVID, according to an NIH press release.
The study included 11,785 participants. At least six months after they were infected with COVID, researchers assessed whether they had developed chronic fatigue. They then compared those results to 1,439 people who did not have COVID.
The researchers determined that cases of ME/CFS were 15 times higher than pre-pandemic levels. (iStock)
“There are no specific tests or biomarkers, so ME/CFS is diagnosed when someone has a constellation of specific symptoms,” Vernon told Fox News Digital.
“This study asked participants questions to determine what symptoms they were experiencing, which can be viewed as a limitation but is currently the approach used to diagnose ME/CFS.”
“It’s well-known that chronic fatigue syndrome can occur in the setting of viral infections.”
Dr. Kenneth J. Perry, a physician based in South Carolina, wasn’t surprised by the incidence of chronic fatigue after a SARS-CoV-2 infection.
“It’s well-known that chronic fatigue syndrome can occur in the setting of viral infections,” Perry, who was not involved in the research, told Fox News Digital.
“Given the fact that SARS-CoV-2 was a novel virus at the time of the pandemic, the incidence and prevalence of such post-infection syndromes was difficult to predict.”
“It’s well-known that chronic fatigue syndrome can occur in the setting of viral infections,” a physician told Fox News Digital. (iStock)
The study does have some limitations when attempting to compare between different viruses, the doctor noted.
“Patients were required to self-report their symptoms,” he said. “This makes the ability to compare symptoms across viruses difficult, as the spotlight on COVID will increase the possibility of selection bias.”
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It’s difficult to specifically determine how chronic fatigue happens in the setting of a viral infection, according to Perry.
“More studies would have to be done to determine the actual underlying pathophysiology of the disease,” he said. “Given that lack of complete understanding, it is difficult to determine how to adequately prevent the syndrome.”
What to know about chronic fatigue
ME/CFS is a “complex, serious and chronic condition that often occurs following an infection,” the NIH wrote.
The condition is marked by “new-onset fatigue” that lasts for at least six months and impairs the person’s ability to engage in ordinary activities.
“There are both physical and psychological components to the syndrome.”
Another symptom is “post-exertional malaise,” in which the person feels extreme fatigue after physical or mental activity, the study stated.
People with ME/CFS may also experience “unrefreshing sleep,” as well as cognitive impairment or dizziness when standing.
Many of these symptoms are also associated with long COVID, the researchers wrote.
“Chronic fatigue syndrome can be very problematic for patients,” Perry told Fox News Digital. “There are both physical and psychological components of the syndrome.”
“Having a relationship with a physician who knows your baseline activity level is going to ensure an appropriate evaluation and intervention if there are any changes,” a doctor advised. (iStock)
Perry reiterated that patients can experience direct fatigue and a decreased ability to do normal activities — and this can also lead to anxiety and depression due to these changes.
“It is very difficult to adequately understand the difference between chronic fatigue syndrome and … underlying psychiatric/psychological health changes,” he said. “This makes it a very difficult disease to explain for patients.”
‘Urgent call to action’
Those experiencing symptoms of chronic fatigue should contact their primary care doctor, Perry advised.
“Having a relationship with a physician who knows your baseline activity level is going to ensure an appropriate evaluation and intervention if there are any changes,” he said.
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“For example, a triathlete who can no longer even stay awake through the entirety of a workday would absolutely be concerning to a physician who knows that patient well. This relationship would also allow for appropriate medications and therapies to be initiated earlier in the process.”
“The dramatic increase in ME/CFS cases post-COVID-19 means that providers will encounter this condition far more frequently,” the researcher said. (iStock)
This research underscores an “urgent call to action,” Vernon said — “especially to healthcare providers.”
“The dramatic increase in ME/CFS cases post-COVID-19 means that providers will encounter this condition far more frequently,” she predicted.
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The researcher called for early recognition and proper management of ME/CFS, which she said can be “life-changing” for patients.
“We hope this study helps everyone understand that ME/CFS is a real and diagnosable condition that demands attention, especially in the wake of a global pandemic.”
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Eating too much salt has long been linked to high blood pressure, but new research suggests it could trick the immune system into prematurely aging the blood vessels.
A preclinical study recently published in the Journal of the American Heart Association has identified a biological chain reaction that links a salty diet to cardiovascular decay.
Scientists at the University of South Alabama observed that mice on a high-salt diet experienced rapid deterioration in their blood vessel function.
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After just four weeks of high sodium intake, the small arteries responsible for regulating blood flow lost their ability to relax, according to a press release.
The team found that the cells lining these vessels had entered a state of cellular senescence, a form of premature cellular aging in which cells stop dividing and release a mix of inflammatory signals that can damage surrounding tissue.
Excess salt has long been linked to high blood pressure, but a new study goes deeper into its effects on the cardiovascular system. (iStock)
The researchers tried to replicate this damage by exposing blood vessel cells directly to salt in a laboratory dish, but the cells showed no harmful effects.
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This suggests that salt isn’t directly causing damage to the vascular lining but that the real culprit may be the body’s own defense mechanism, the researchers noted.
Excess salt may trigger the immune system to release a molecule called interleukin-16 (IL-16), which acts as a messenger that instructs blood vessel cells to grow old before their time, according to the study.
Excess salt may trigger the immune system to release a molecule called interleukin-16, which acts as a messenger that instructs blood vessel cells to grow old before their time, according to the study. (iStock)
Once these cells age, they fail to produce nitric oxide, the essential gas that tells arteries to dilate and stay flexible.
To test whether this process could be reversed, the team turned to a class of experimental drugs known as senolytics.
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Using a cancer medication called navitoclax, which selectively clears out aged and dysfunctional cells, the researchers were able to restore nearly normal blood vessel function in the salt-fed mice, the release stated.
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By removing the decaying cells created by the high-salt diet, the drug allowed the remaining healthy tissue to maintain its elasticity and respond correctly to blood flow demands.
Excess salt may trigger the immune system into stopping the cells from dividing, the study suggests. (iStock)
The study did have some limitations. The transition from mouse models to human treatment remains a significant hurdle, the team cautioned.
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Senolytic drugs like navitoclax are still being studied for safety, and the team emphasized that previous trials have shown mixed results regarding their impact on artery plaque.
Additionally, the researchers have not yet confirmed whether the same IL-16 pathway is the primary driver of vascular aging in humans.
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Eating a diet high in fruits and vegetables was found to have a surprising link to lung cancer among younger non-smokers, early research suggests.
The observational study, led by Jorge Nieva, M.D., of the USC Norris Comprehensive Cancer Center at Keck Medicine, was presented this month at the American Association for Cancer Research (AACR) annual meeting in San Diego. It has not yet been peer-reviewed.
Researchers looked at dietary, smoking and demographic data for 187 patients who were diagnosed with lung cancer at age 50 or younger.
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They found that among non-smokers, there was a link between healthier-than-average diets – rich in fruits, vegetables and whole grains – and the chance of lung cancer development.
Young lung cancer patients ate more servings of dark green vegetables, legumes and whole grains compared to the average U.S. adult, the researchers found.
Eating a diet high in fruits and vegetables was found to have a surprising link to lung cancer among younger non-smokers, early research suggests. (iStock)
The researchers hypothesized that pesticides applied to conventionally grown produce could be a possible factor in the disease association.
“Commercially produced (non-organic) fruits, vegetables and whole grains are more likely to be associated with a higher residue of pesticides than dairy, meat and many processed foods,” according to Nieva. He also noted that agricultural workers exposed to pesticides tend to have higher rates of lung cancer.
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“There is a large subset of lung cancer patients whose disease is not caused by smoking,” Nieva told Fox News Digital.
The disease is becoming more common in non-smokers 50 and younger, especially women – despite the fact that smoking rates have been falling for decades, the researcher noted.
The researchers hypothesized that pesticides applied to conventionally grown produce could be a possible factor in the disease association. (iStock)
“These patients tend to have eaten much healthier diets before their diagnosis than the average American,” he went on. “We need to support research into understanding why Americans – and women in particular – who no longer smoke very much are still having lung cancer,” he said.
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The study did have some limitations, Nieva acknowledged, primarily that it relied on survey data and was limited by the participants’ memories of their food intake.
“Also, the survey participants were self-selected, and this could have biased the findings,” he told Fox News Digital.
“There is a large subset of lung cancer patients whose disease is not caused by smoking.”
The researchers did not test specific foods for pesticides, relying instead on average pesticide levels for certain types of food. Looking ahead, they plan to test patients’ blood and urine samples to directly measure pesticide levels, Nieva said.
Although the study shows only an association and does not prove that pesticides caused lung cancer, Nieva recommends that people wash their produce before eating and choose organic foods whenever possible.
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“This work represents a critical step toward identifying modifiable environmental factors that may contribute to lung cancer in young adults,” said Nieva. “Our hope is that these insights can guide both public health recommendations and future investigation into lung cancer prevention.”
“It is possible that the increased lung cancer risk could be due to pesticide exposure in whole farmed foods, but is by no means certain,” a doctor said. (AP Photo/Charlie Neibergall)
Dr. Marc Siegel, Fox News senior medical analyst, said the study is “interesting,” but that it “raises far more questions than it answers.”
“It is a small study (around 150) and observational, so no proof,” the doctor, who was not involved in the research, told Fox News Digital.
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“It is possible that the increased lung cancer risk could be due to pesticide exposure in whole farmed foods, but it is by no means certain,” Siegel went on. “How much exposure is needed? How much of it gets into food and in which areas? This requires much further study.”
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Kayla Nichols, communications director for Pesticide Action & Agroecology Network, a distributed global network, said the organization agrees with the study’s conclusion that more research should be done on the rise in lung cancer, particularly in individuals eating diets higher in produce and fiber.
“There is a large subset of lung cancer patients whose disease is not caused by smoking,” the researcher told Fox News Digital. (iStock)
“There is a bounty of existing research that already links pesticide exposure to increased risk of multiple types of cancers,” Nichols, who was also not involved in the study, told Fox News Digital. She called for more research on chronic, low-level exposures to pesticides, as well as more effective policies to protect the public from pesticide residues on food.
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The study was supported by the National Institutes of Health and the National Cancer Institute, as well as industry partners including AstraZeneca and Genentech, among others.
Fox News Digital reached out to several pesticide companies and trade groups for comment.
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