Health
Hantavirus fears spark COVID flashbacks, but experts say there’s one major difference
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Concerns about rising hantavirus cases has Americans reflecting on the coronavirus pandemic.
Although COVID-19 began with a foreign strain and spread rapidly around the world, experts say it’s not likely that hantavirus will behave the same way.
The rare Andes virus, which was linked to the MV Hondius cruise ship outbreak, is the only known hantavirus strain that has the capability to spread from person to person, usually through prolonged close contact.
RARE HANTAVIRUS HUMAN-TO-HUMAN TRANSMISSION SUSPECTED ON LUXURY CRUISE SHIP WHERE 3 HAVE DIED
Fox News senior medical analyst Dr. Marc Siegel spoke with Fox News Digital about the similarities and differences between hantavirus and coronavirus, noting that there is “no comparison.”
“You could say the comparison ends at that they’re both single-stranded RNA viruses,” he said. “That’s a comparison, but [hantavirus] has been unchanged basically for decades.”
Dr. Marc Siegel says there’s “no comparison between these two viruses, other than that the single-stranded RNA viruses are both carried by animals.” (iStock)
Coronavirus was different because it began to mutate, which started to cause “all kinds of problems,” Siegel noted.
“We don’t know why it started to mutate, but this one doesn’t appear to have done that,” he said. “And every day that goes by seems to show that theory is correct – the genetics of it is the same.”
DR MARC SIEGEL: HANTAVIRUS CRUISE OUTBREAK IS ALARMING BUT FEAR IS SPREADING FASTER THAN FACTS
“So, there’s no comparison between these two viruses, other than that the single-stranded RNA viruses are both carried by animals.”
Siegel added that COVID is an airborne virus, while hantavirus is mainly a secretion-borne virus, although it can be transmitted through dust and droppings in the air.
The rare Andes virus, which was linked to the MV Hondius cruise ship outbreak, is the only known hantavirus strain that has the capability to spread from person to person, usually through prolonged close contact. (Andres Gutierrez/Anadolu)
“It’s not airborne … in terms of respiratory droplets hanging in the air,” he said. “It’s very difficult to transmit.”
While coronavirus “moved in the direction of humans in a significant way,” hantavirus has not, except for “very rare” human-to-human transmission, according to the doctor.
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There have been hantavirus cases in the U.S. for decades, although they are “very rare,” Siegel noted.
Certain factors of this disease spread are changing, including warming temperatures that are causing rodents to migrate north toward Buenos Aires, according to the doctor.
DEADLY CRUISE VIRUS TIMELINE SHOWS HOW HANTAVIRUS SPREAD AMONG PASSENGERS
The current outbreak stemming from the cruise ship did not help the cause, Siegel went on — but this spread doesn’t suggest that the virus has changed. Rather, it shows how close quarters on a ship are “very conducive” to spread, he said.
Passengers disembark from the hantavirus-stricken cruise ship MV Hondius at the port of Granadilla in Tenerife, Canary Islands, Spain, on May 10, 2026. (AP Photo)
“Every day that goes by shows that … we’re not seeing a second generation of spread,” he reiterated.
The better comparison to make is between hantavirus and bird flu, which is a predominantly animal-based virus that “occasionally infects humans,” Siegel said.
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“There are billions of birds, and every year we talk about how it’s going to cause a pandemic, but it would have to mutate significantly,” he pointed out. “I feel that [hantavirus] would have to mutate significantly before it could go human to human in any significant way, because this is basically an animal virus … it’s very comfortable inside a rodent host.”
Siegel went on, “If you get this virus, you’re in trouble, but getting this virus is very difficult.”
A person visits a COVID testing site on a Manhattan street in New York City on Jan. 21, 2022. “Coronaviruses are airborne … this is not,” Dr. Siegel said. “And coronaviruses mutate a lot, and this does not.” (Spencer Platt/Getty Images)
Regarding fears that another global pandemic may be looming, Siegel said that just because one virus becomes widespread does not mean all viruses will.
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“Coronaviruses are airborne anyway. This is not. And coronaviruses mutate a lot, and this does not,” he said. “I’m much more concerned about flu than this. Flu can mutate all the time, and it’s already going human to human all over the place, and it’s airborne.”
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“Most infectious disease specialists are much more worried about flu than this, as deadly as this can be,” he added.
“We’re talking apples and oranges, and any comparison you make after that provokes fear.”
Fox News Digital’s Melissa Rudy contributed to this report.
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An accidental lab discovery has opened the door to entirely new ways of preventing the flu.
While investigating how influenza replicates, researchers discovered that different flu strains use completely different strategies to infiltrate human cells, SWNS reported.
By targeting the specific molecules the viruses rely on, scientists found that they could block them from entering new cells and halt their replication altogether.
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Researchers say these “fundamental insights” into seasonal influenza highlight a clear path toward developing better preventive medications.
“The hope is that fundamental, curiosity-based research like this helps to pave the way for novel strategies to treat and prevent influenza infections,” principal investigator Dr. Emily Bruce, from the University of Vermont’s Larner College of Medicine, said in the SWNS report.
While investigating how influenza replicates, researchers discovered that different flu strains use completely different strategies to infiltrate human cells. (iStock)
While several flu strains cause illness, H1N1 and H3N2 influenza A viruses are the most common. However, current flu tests cannot differentiate between them, and clinical treatments are identical for both.
Although vaccines and antivirals are available, Bruce noted a “dire” need for better medications to stop the virus from spreading cell to xxcell.
“You don’t get sick when a virus is in one cell,” he noted. “You get sick because a virus replicates itself and goes into many more cells.”
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The study, which was published in The Journal of Virology, originally aimed to map how viral RNA segments are transported within cells to create new viral particles.
The team used H1N1 and H3N2 viruses isolated from the nasal passages of positive patients in 2022.
Clinical treatments remain identical for both primary strains of the flu virus. (iStock)
During the investigation, the team unexpectedly stumbled upon a cellular pathway that blocked the virus from entering lung cells, SWNS reported.
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The data revealed that when a specific human protein called Rab11B was depleted, H3N2 viruses failed to enter human lung cells. H1N1 viruses were completely unaffected.
Using reverse genetics, the team mapped this defect and uncovered a brand-new, H3N2-specific role for Rab11B during viral entry.
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This discovery challenged the scientific assumption that all flu viruses enter cells the same way.
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“Viruses are like pirates from different countries hijacking someone’s ship,” Bruce said. “Different viruses, like different types of pirates, use different methods to get onboard.”
This discovery challenged the scientific assumption that all flu viruses enter cells the same way. (iStock)
“We had previously thought that all flu viruses used the same way to get into a cell, but we discovered that this is not true,” she went on. “H1N1 and H3N2 need different proteins to get in, and if you get rid of the right protein, a specific virus can’t get in.”
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While these findings identify a critical cellular pathway for viral entry, the study was conducted using isolated cells, the researchers acknowledged.
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Further research is needed to determine whether blocking the protein is safe and effective within a live, complex human respiratory system.
Bruce and the team hope to conduct further research to determine whether this Rab11B-dependency is a fundamental property of H3N2, or if it’s a trait unique to currently circulating flu strains.
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Eating processed meat like ham, sausage and bacon may be linked to a higher risk of certain types of cancer, according to new research.
While health organizations have already confirmed that processed meat can contribute to colon cancer, this study looked closer at cancers in the upper digestive tract, where the link has historically been less clear.
To understand these connections, researchers from the European Prospective Investigation into Cancer and Nutrition (EPIC), one of the world’s largest long-term nutrition and cancer cohorts, tracked the health and diets of 450,112 people across Europe for an average of 14 years.
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The study group included 131,426 men and 318,686 women, according to the study’s press release.
During the follow-up period, 876 people developed stomach cancer and 215 people developed esophageal adenocarcinoma, which is cancer of the tube connecting the mouth to the stomach.
For female participants, eating both processed meat and white meat was linked to an increased risk of developing the disease. (iStock)
Researchers tracked where the stomach cancers grew, separating them into the upper part of the stomach near the throat and the lower part of the stomach.
The researchers also sorted the tumors into two categories based on how the cancer cells appeared under a microscope: intestinal, which forms more organized structures, and diffuse, in which the cells are more scattered throughout the tissue.
BACTERIA IN YOUR MOUTH MAY TRAVEL TO THE GUT AND TRIGGER STOMACH CANCER, RESEARCH FINDS
After adjusting for other lifestyle factors, the researchers found that for every extra 30 grams of processed meat a person ate per day, their overall risk of stomach cancer went up by 9%. Eating that same extra 30 grams a day was also linked to a 13% higher risk of esophageal adenocarcinoma.
A standard single slice of regular deli-sliced ham or lunch meat averages around 28 grams, according to USDA data and nutritional tracking databases.
An extra 20 grams of white meat, such as chicken and turkey, was linked to a 12% higher risk of cancer in the main body of the stomach. (iStock)
An extra 20 grams of white meat, such as chicken or turkey, was linked to a 12% higher risk of cancer in the main body of the stomach, the researchers noted.
The study also revealed differences between men and women. For male participants, only processed meat showed a clear, statistically significant link to a higher risk of stomach cancer. For female participants, however, eating both processed meat and white meat was linked to an increased risk.
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These findings align with global health benchmarks, particularly those established by the World Health Organization’s International Agency for Research on Cancer.
The agency has long classified processed meat as a known human carcinogen, primarily due to its strong, well-documented links to colorectal cancer.
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However, health organizations have also consistently pointed to a potential, yet less definitive, relationship between these meats and cancers of the stomach.
Eating 30 grams of processed meat a day, or the equivalent to one slice of ham, was linked to a 13% higher risk of esophageal adenocarcinoma. (iStock)
Further scientific investigation is needed to confirm the findings and to account for other underlying risk factors, such as certain stomach infections, which could interact with dietary habits.
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A key limitation of the study is its reliance on self-reported diets, which can sometimes lead to inaccuracies in how participants recall their meat consumption over time, the researchers noted.
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The findings were published in the International Journal of Cancer.
Fox News Digital reached out to the researchers requesting comment.
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