A newborn baby in New Mexico died from a Listeria infection likely tied to the mother drinking raw milk while pregnant, according to state health officials.

The New Mexico Department of Health issued a warning Tuesday urging people to avoid consuming raw dairy products following the newborn’s death. Health officials believe the “most likely” source of infection was the mother drinking unpasteurized milk during pregnancy.

While investigators said they could not determine the exact cause, they said the “tragic death underscores the serious risks raw dairy poses to pregnant women, young children, elderly New Mexicans and anyone with a weakened immune system.”

Raw milk has seen a surge in popularity amid the Make America Healthy Again movement led by Health and Human Services Secretary Robert F. Kennedy Jr.

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The U.S. Food and Drug Administration issued a regulation decades ago prohibiting the interstate sale of raw milk, but the drink is not federally banned, leaving individual states to decide whether it’s safe for human consumption.

“Individuals who are pregnant should only consume pasteurized milk products to help prevent illnesses and deaths in newborns,” Dr. Chad Smelser, deputy state epidemiologist for the New Mexico Department of Health said in a statement.

Raw milk has not been pasteurized — a process that heats milk to remove disease-causing germs.

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Consuming food or beverages made from raw milk can expose people to Campylobacter, Cryptosporidium, E. coli, Listeria, Brucella and Salmonella, according to the Centers for Disease Control and Prevention (CDC).

Listeria is the third leading cause of death from foodborne illness in the U.S., infecting about 1,250 people and causing roughly 172 deaths each year, according to the CDC.

The CDC notes that certain groups of people, including children under the age of 5, adults over 65, pregnant women and people with weakened immune systems, have a higher risk of serious illness.

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“New Mexico’s dairy producers work hard to provide safe, wholesome products and pasteurization is a vital part of that process,” Jeff M. Witte, New Mexico secretary of agriculture, said in a statement. “Consumers, particularly those at higher risk, are encouraged to choose pasteurized dairy products to reduce the risk of serious foodborne illness.”

Last August, an outbreak of E. coli and Campylobacter linked to raw milk from a Florida farm sickened 21 people, including six children.

As marijuana’s potential health benefits are under debate, new research has found that THC could have a powerful and positive effect on the brain.

Researchers at the University of Texas (UT) at San Antonio, Long School of Medicine, recently discovered that THC (tetrahydrocannabinol, the main psychoactive compound in marijuana) could prevent the development of Alzheimer’s disease – but only when paired with an anti-inflammatory drug called celecoxib.

Celecoxib is a selective COX-2 inhibitor that is commonly prescribed for arthritis and pain.

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While THC has previously been shown to have anti-inflammatory and neuroprotective properties, it’s also been linked to negative effects on the brain, particularly in learning and memory, according to a UT Health press release.

Combining THC with an anti-inflammatory drug, however, could provide the same benefits with fewer negative side effects.

The study, which was published in the journal Aging and Disease, paired low-dose THC extract with celecoxib, which was administered to mice daily for 30 days.

The mice received the treatments before the development of memory symptoms to measure the impact on preventing or delaying Alzheimer’s.

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The results showed improved cognition, learning and memory, as well as decreased markers of neuroinflammation and reduced Alzheimer’s-related brain pathology, the release stated.

Although THC alone had the same results, it also increased inflammatory signals, while the combination dosage did not.

“What really mattered was behavior. If cognition is not improved, then the treatment doesn’t matter. And that’s where the combination clearly worked better than THC alone,” noted lead study author Chu Chen, Ph.D., professor in the Department of Cellular and Integrative Physiology.

Years of research led Chen to connect the dots on how THC can manipulate the brain and why it’s difficult to use safely for neurological conditions, according to UT Health.

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“When THC is given, it unexpectedly increases COX-2 in the brain. That increase is closely associated with learning and memory impairment,” Chen said.

Both THC and celecoxib are FDA-approved for use in humans, which could provide a “real advantage” in moving along to clinical trials, according to UT Health.

“What really mattered was behavior. If cognition is not improved, then the treatment doesn’t matter.”

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Future studies will investigate whether the drug combination can slow disease progression or reverse deficits after symptoms have appeared.

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“This work has taken many years. But now we’re at a point where basic neuroscience discoveries are pointing toward something that could realistically move into the clinic,” Chen said.

Dr. Paul Saphier, a neurosurgeon and founder of Coaxial Neurosurgical Specialists in New Jersey, called this research an “exciting breakthrough with a huge potential impact for patients and their families.”

“Neurocognitive deterioration has been linked to conditions that cause inflammation within the brain — prior brain bleeds, strokes, tumors and infections,” he told Fox News Digital. “We also know that the buildup of the plaques related to Alzheimer’s [beta-amyloid plaques] are the result of chronic inflammation.”

“So, it makes sense that this combination therapy [might] help limit the production of this amyloid and thereby reduce the progression of the disease.”

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Saphier said the drugs’ FDA approval status is “encouraging,” as it may “ultimately ease the availability for patients, if the trial proves successful.”

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“I look forward to the results of this trial, as well as any future trials that look at limiting the effects of chronic inflammation within the central nervous system.”

Puzzling cancer and Alzheimer’s link explored in new study: ‘SHORT QUOTE’

A new study may help clarify a long-observed link between cancer history and reduced Alzheimer’s risk.

The research, published in the journal Cell, used mouse models to explore how the biological changes associated with cancer may affect the progression of the most common dementia type.

The findings suggest that certain cancers release a protein called cystatin-C that can travel through the bloodstream and enter the brain, an area that many Alzheimer’s treatments have difficulty reaching.

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Cystatin-C binds to harmful amyloid protein clumps linked to Alzheimer’s and activates a protein called TREM2, which acts like an on-switch for the brain’s immune cells. 

When activated, those cells begin clearing away existing amyloid plaques, a process that was associated with reduced plaque buildup and improved cognitive function, the study found.

Dr. Bob Arnot, a Vermont-based internal medicine physician, told Fox News Digital that the findings help clarify a pattern doctors have observed for years.

“Scientists have long observed a puzzling statistical pattern known as ‘inverse comorbidity’ — people with a history of cancer are less likely to develop Alzheimer’s disease, and people with Alzheimer’s are less likely to develop cancer,” said Arnot, who was not involved in the study.

The findings suggest that biological signals tied to certain cancers may slow or reverse key features of Alzheimer’s disease.

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Arnot said the research is notable because it identifies a specific biological pathway that could be targeted for future Alzheimer’s treatments.

“This approach targets existing amyloid plaques, not just early prevention. That distinction could be critical for patients who already have established disease,” he said.

Arnot cautioned that the findings do not mean cancer itself is protective.

“This study does not suggest that cancer is protective, desirable or a viable therapy,” he said. “Instead, it reveals that biological programs activated during cancer can inadvertently engage protective immune mechanisms in the brain.”

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The research suggests that future treatments could focus not just on slowing Alzheimer’s, but also on clearing harmful buildup in the brain, Arnot added.

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The results were based on experiments in animal models, and further research is needed to determine whether the same effects occur in humans, the researchers noted.