Health
Former MLB pitcher finds liver donor in high-school classmate he hadn't seen in 20 years
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A man’s Facebook plea for a liver donor attracted the attention of a high-school acquaintance he hadn’t spoken to in 20 years.
In April 2024, after experiencing appetite loss and losing 15 pounds in a month, Steven Register, 42, was diagnosed with stage 4 colon cancer, according to news agency SWNS.
Doctors told the former MLB pitcher — who played for the Colorado Rockies in 2008 and the Philadelphia Phillies in 2009 — that a liver transplant was likely his best chance of survival.
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“When we first met with the doctors, they gave me a year to a year-and-a-half to live,” Register said, adding that he and his wife, Beth, immediately started researching options.
The couple traveled from Auburn, Alabama, to the MD Anderson Cancer Center in Houston, Texas, where Register underwent surgery for a temporary ostomy bag and began chemotherapy, per the SWNS report.
“When we first met with the doctors, they gave me a year to a year-and-a-half to live,” said Steven Register, shown above with his wife, Beth. (SWNS)
Plans for the liver resection were canceled when doctors discovered the tumors were too large, which led the couple to consider a transplant.
Register’s wife created a Facebook group to search for a living liver donor, hoping someone would come forward in time.
An unexpected volunteer
Kristin Johnston, a 40-year-old preschool teacher from Roswell, Georgia, saw the post and recognized Register as a former high-school classmate.
The two had met in 1999 at Shaw High School in Columbus, Georgia, but hadn’t spoken in over two decades.
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“It was just so far out of left field,” said Register, who couldn’t believe it when Johnston volunteered to donate part of her liver.
He added to SWNS, “I haven’t seen or talked to her in over 20 years, and for her to reach out like that, it was just meant to be.”
Kristin Johnston, a 40-year-old preschool teacher from Roswell, Georgia, shown above, saw Register’s Facebook post and recognized him as a former high-school classmate. (SWNS)
Johnston said she started by doing a quick online search for live liver donation and discovered that blood type compatibility was the first step.
“I just sent him a message,” she said. “I said, ‘Hey, what’s your blood type?’ and he said, ‘I’m B positive.’”
She responded, “Wait, that’s mine, too,” and offered, “I’ll happily donate a lobe if I’m a match.”
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Johnston was later cleared as a match and received the confirmation on a meaningful day.
“I got the news on Good Friday, of all days, that I was officially cleared as his liver donor,” she said.
Surgery and second chances
The surgery, which is expected to take 12 to 14 hours, will involve removing 70% of Johnston’s liver and transplanting it into Register, according to SWNS.
Both her remaining liver and the donated portion will regenerate over time, giving both a second chance at health.
“I got the news on Good Friday, of all days.”
“For her, she is ultimately giving him the gift of life — for him, a really fresh start in this journey,” said Beth Register.
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Steven Register said he hoped that “once I get my liver with all the tumor and cancer out, I’ll be cancer-free from there.”
The Register family — including children McKenzie (16), Blakely (14) and Brooks (8) — launched a fundraiser on SupportNow to help with travel, food and medical expenses.
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Johnston and her husband Cody, 38, a real estate developer, have three kids of their own — Sawyer (9), Teddy (7) and Dahlia (4).
Raising awareness and faith
Beth Register said she hopes their family’s story raises awareness about the impact of living organ donation.
“A lot of people don’t realize that live donations, whether it be for livers or kidneys, are even an option,” she said.
The Register family — Steven, Beth and their three children, McKenzie (middle-left), Blakely (middle-right) and Brooks (front, center) — launched a fundraiser on SupportNow to help with travel, food and medical expenses. (SWNS)
She added that Johnston had been selfless from the start.
The families believe fate had a hand in reconnecting them, per SWNS.
“We just pray that God is opening all the right doors and that Kristin is the perfect donor for him.”
“We just pray that God is opening all the right doors and that Kristin is the perfect donor for him,” Beth Register said.
“We just appreciate her being willing to put her life on pause to hopefully lengthen his life by many, many, many years.”
Health
New ways to prevent flu revealed in ‘accidental’ lab breakthrough, study finds
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An accidental lab discovery has opened the door to entirely new ways of preventing the flu.
While investigating how influenza replicates, researchers discovered that different flu strains use completely different strategies to infiltrate human cells, SWNS reported.
By targeting the specific molecules the viruses rely on, scientists found that they could block them from entering new cells and halt their replication altogether.
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Researchers say these “fundamental insights” into seasonal influenza highlight a clear path toward developing better preventive medications.
“The hope is that fundamental, curiosity-based research like this helps to pave the way for novel strategies to treat and prevent influenza infections,” principal investigator Dr. Emily Bruce, from the University of Vermont’s Larner College of Medicine, said in the SWNS report.
While investigating how influenza replicates, researchers discovered that different flu strains use completely different strategies to infiltrate human cells. (iStock)
While several flu strains cause illness, H1N1 and H3N2 influenza A viruses are the most common. However, current flu tests cannot differentiate between them, and clinical treatments are identical for both.
Although vaccines and antivirals are available, Bruce noted a “dire” need for better medications to stop the virus from spreading cell to xxcell.
“You don’t get sick when a virus is in one cell,” he noted. “You get sick because a virus replicates itself and goes into many more cells.”
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The study, which was published in The Journal of Virology, originally aimed to map how viral RNA segments are transported within cells to create new viral particles.
The team used H1N1 and H3N2 viruses isolated from the nasal passages of positive patients in 2022.
Clinical treatments remain identical for both primary strains of the flu virus. (iStock)
During the investigation, the team unexpectedly stumbled upon a cellular pathway that blocked the virus from entering lung cells, SWNS reported.
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The data revealed that when a specific human protein called Rab11B was depleted, H3N2 viruses failed to enter human lung cells. H1N1 viruses were completely unaffected.
Using reverse genetics, the team mapped this defect and uncovered a brand-new, H3N2-specific role for Rab11B during viral entry.
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This discovery challenged the scientific assumption that all flu viruses enter cells the same way.
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“Viruses are like pirates from different countries hijacking someone’s ship,” Bruce said. “Different viruses, like different types of pirates, use different methods to get onboard.”
This discovery challenged the scientific assumption that all flu viruses enter cells the same way. (iStock)
“We had previously thought that all flu viruses used the same way to get into a cell, but we discovered that this is not true,” she went on. “H1N1 and H3N2 need different proteins to get in, and if you get rid of the right protein, a specific virus can’t get in.”
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While these findings identify a critical cellular pathway for viral entry, the study was conducted using isolated cells, the researchers acknowledged.
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Further research is needed to determine whether blocking the protein is safe and effective within a live, complex human respiratory system.
Bruce and the team hope to conduct further research to determine whether this Rab11B-dependency is a fundamental property of H3N2, or if it’s a trait unique to currently circulating flu strains.
Health
One extra serving of processed meat a day linked to higher cancer risk
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Eating processed meat like ham, sausage and bacon may be linked to a higher risk of certain types of cancer, according to new research.
While health organizations have already confirmed that processed meat can contribute to colon cancer, this study looked closer at cancers in the upper digestive tract, where the link has historically been less clear.
To understand these connections, researchers from the European Prospective Investigation into Cancer and Nutrition (EPIC), one of the world’s largest long-term nutrition and cancer cohorts, tracked the health and diets of 450,112 people across Europe for an average of 14 years.
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The study group included 131,426 men and 318,686 women, according to the study’s press release.
During the follow-up period, 876 people developed stomach cancer and 215 people developed esophageal adenocarcinoma, which is cancer of the tube connecting the mouth to the stomach.
For female participants, eating both processed meat and white meat was linked to an increased risk of developing the disease. (iStock)
Researchers tracked where the stomach cancers grew, separating them into the upper part of the stomach near the throat and the lower part of the stomach.
The researchers also sorted the tumors into two categories based on how the cancer cells appeared under a microscope: intestinal, which forms more organized structures, and diffuse, in which the cells are more scattered throughout the tissue.
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After adjusting for other lifestyle factors, the researchers found that for every extra 30 grams of processed meat a person ate per day, their overall risk of stomach cancer went up by 9%. Eating that same extra 30 grams a day was also linked to a 13% higher risk of esophageal adenocarcinoma.
A standard single slice of regular deli-sliced ham or lunch meat averages around 28 grams, according to USDA data and nutritional tracking databases.
An extra 20 grams of white meat, such as chicken and turkey, was linked to a 12% higher risk of cancer in the main body of the stomach. (iStock)
An extra 20 grams of white meat, such as chicken or turkey, was linked to a 12% higher risk of cancer in the main body of the stomach, the researchers noted.
The study also revealed differences between men and women. For male participants, only processed meat showed a clear, statistically significant link to a higher risk of stomach cancer. For female participants, however, eating both processed meat and white meat was linked to an increased risk.
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These findings align with global health benchmarks, particularly those established by the World Health Organization’s International Agency for Research on Cancer.
The agency has long classified processed meat as a known human carcinogen, primarily due to its strong, well-documented links to colorectal cancer.
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However, health organizations have also consistently pointed to a potential, yet less definitive, relationship between these meats and cancers of the stomach.
Eating 30 grams of processed meat a day, or the equivalent to one slice of ham, was linked to a 13% higher risk of esophageal adenocarcinoma. (iStock)
Further scientific investigation is needed to confirm the findings and to account for other underlying risk factors, such as certain stomach infections, which could interact with dietary habits.
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A key limitation of the study is its reliance on self-reported diets, which can sometimes lead to inaccuracies in how participants recall their meat consumption over time, the researchers noted.
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The findings were published in the International Journal of Cancer.
Fox News Digital reached out to the researchers requesting comment.
Health
The Surprising Hormone That Could Make Menopause Weight Loss Easier
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